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The pathogenesis of acute tubular necrosis


, Angiotensin converting enzyme inhibitor captopril only early, mild protective effect of acute renal failure, no significant effect of imprison for this view was expressed that, TGF in the pathogenesis of acute renal failure did not play a key role only in the solid-primary disease with acute renal failure caused by renal tubular injury, re-absorption capacity of filtration material circumstances. Compensatory response resulting from a kidney. 2.3 cellular energy metabolism and found a large number of acute renal failure, kidney reflect blood, intracellular ATP content in renal tissue decreased rapidly when the kidney after reperfusion,[link widoczny dla zalogowanych], ATP content can be partially restored,[link widoczny dla zalogowanych], and the speed of recovery time of ischemic directly related. Recently, studies suggest that renal ischemia Zd 'time after. GFR and directly related to intracellular ATP content. Another study found that many in the ischemic and toxic acute renal failure early and kidney cell ATP, ADP levels have decreased, while AMP, inorganic phosphate levels. Ischemic acute renal failure in animals to exogenous ATP, can significantly improve cell morphology and function. Independent of its role and vascular function. Inhibitor of adenosine removal by maintaining cellular ATP, can significantly improve ischemic acute renal failure renal function recovery. Because many of the basic cells or special function or role is ATP Nong Lai nature. Therefore, decreased intracellular ATP levels can cause cell injury and death of another. AMP levels decreased after ATP, and AMP can be generated by further metabolism of adenosine, sub-xanthine brick, brick xanthine, oxygen free radicals, which are considered fixed factors involved in the pathogenesis of acute renal failure. And acute renal failure 2.4 Adenosine Adenosine is a metabolite of adenosine in the hypoxic tissues have increased significantly. It was found that adenosine can cause GFR and filtration fraction marked and continuous decline, and its role in acute renal failure and renal hemodynamic changes are very similar; and also by renal arterial infusion of adenosine receptor antagonist theophylline, will enable the missing bloody animals with acute renal failure was significantly higher GFR dipyridamole can prevent the cells to adenosine uptake, thereby increasing extracellular adenosine concentrations, ischemic acute renal failure in animals given intravenous administration of dipyridamole, can significantly increase the GFR, RPF and filtration fraction decreased. Another study showed that theophylline on glycerol, acute renal failure caused by endotoxin is also part of the protective effect, so that GFR, RPF celebration Trappe increase in claims, however, mercuric chloride induced acute renal failure in Sui, adenosine showed no significant role. Intermediate to Journal Volume 31 96 5 2,6 main blue emergency. . Often without pole pole!!: Zhu limonene very thin Chi Man '; due; can be high _ the public. Multi Zhu '÷ limonene Zhu Qian Zhu! _N. Glycosaminoglycan analogy Zhu hand off drug; decline; Io1 please =,, cylinder cell luminal membrane and the basolateral membrane due to different functions. A drunk in the luminal membrane, Liu. : 'Quiet, and transport protein related substances may be co-Hang related materials prepared from the f of small children. Lan Lilan necrosis is, in former times when the ischemic acute renal proximal tubular epithelial cells the basolateral membrane. : Eleven - an ugly one on the H transferred to the luminal membrane KATP activity,[link widoczny dla zalogowanych], luminal membrane Na-K-ATP activity of a quantity and ischemic time of the ancient parallel. The luminal membrane lipid composition of basolateral membrane protein distribution has also changed, phospholipids and bile imprison the two alcohols can be two-way movement between the cell membrane, leading to the basolateral membrane, luminal membrane function and structure to maintain the differences Go to the loss of cell polarity of luminal membrane Na-KATP enzyme can be epithelial cells, Na pumps to the small lumen, resulting in a proximal tubule reabsorption of a barrier,[link widoczny dla zalogowanych], {recovery until the proximal tubule cell polarity is restored,[link widoczny dla zalogowanych], features a small collection tube before resuming heavy flavor; lipid composition and protein distribution changes can be restored during the recovery period. The research suggests. Loss of renal tubular epithelial cell polarity in the pathogenesis of acute renal failure from ~ given role, but its exact position remains to be further studied. 2.7 Endothelin and acute renal failure, endothelin (ET) as appropriate for the University of intravascular West Yu Yu is the first hospital f610041) in recent years. The literature of acute tubular necrosis in the back almost pathological loyalty questioned. That the clinical and experimental acute renal failure, no significant renal tubular epithelial cell necrosis, urine epithelial cells shedding about 30 are alive. The exact pathological mechanism may be related to dysfunction and cell adhesion, which has aroused the attention of its research trends; acute renal failure of different etiology, clinical manifestations, many differences, the light catching a mild oliguria or renal dysfunction, In severe cases the symptoms worse. And the need to boil clean dialysis. Patients have prodromal symptoms big. Such as shock, trauma-induced hypotension, drug or poison intoxication symptoms, some primary glomerular diseases, tubulointerstitial diseases buildings can be associated with renal failure, a few cases no obvious causes. Ignorant of the original foundation of clinical signs of disease, more than a prodrome. Lasted several days or l ~ 0, a
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