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tory burch outlet γ- interferon on human cholangi

 
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PostPosted: Sun 11:18, 13 Mar 2011    Post subject: tory burch outlet γ- interferon on human cholangi

γ- interferon on human cholangiocarcinoma cell Fas / FasL system of regulation of


Contribute to its reduced effective immune escape FasL expression. Specific mechanism is unknown,[link widoczny dla zalogowanych], simply because IFN. The role of Fas upregulation making changes to this amount , or in the Fas / FasL pathway plays a role in signaling ? This needs further study . Que et al reported that Fas table cholangiocarcinoma cell function is defective ,[link widoczny dla zalogowanych], and FasL is a normal function . We found that the expression of QBC939 cell Fas / FasL is a function . And IFN. 7 pairs of the adjustment ,[link widoczny dla zalogowanych], and make their co- culture induced apoptosis in Jurkat cells the ability to decline. Thus this aspect further experimental and clinical studies,[link widoczny dla zalogowanych], may be immune to the treatment of cholangiocarcinoma open up a new way to look to improve the current status of biliary tract cancer with poor prognosis ,[link widoczny dla zalogowanych], with a real clinical significance. ( see insert this figure 8,9 7-1)
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