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Plasma protein C, protein S in patients with venous thrombosis diagnosis and treatment of


. 2001,104 (1): l ~ 618MeijerJC,[link widoczny dla zalogowanych], OtMijkEJ, Mesnier1.0. eta1. ReducedactivityofTAFI (thrombin-activatablefibrlnolysisinhibitor) inacutelm ~ telocrticleukemialJ】. BrJHaemato1.2000, 108 (3): 518 ~ 52319KlementP, LiaoP, BBjzarL. Anovelapproachtoarterialysis】 【J. Blood. 1.099,94 (: 2735 ~ 274320NagashimaM, WemerM, WangM, eta1. Aninhibitor0factivatedthrombin-activatablefibrinolysisinhibitorpotentiatestisane-typeplasmino-genactivator-inducedthrambolysisinarabbitju Head Lllarveint} lIDmb0ly3ismodel [J】. . l ~ ombRes. 2O00, 98 (4): 333 ~ 34221MattssenC, BjorkmanJA, AbrahamssonT, eta1. proCI ~ (TAFI) activationduringtlu-ombolytien coffee ntinadogmodel0fcorenaryarterythrombosiscanbeinhibitedwithadirect. smallmoleculethnm ~ inin-hibitor (mdagatran)】 【J. ThwmbH head fl0st. 2OO2.87 (4) :557-562 (Received :2003-08-25 Revised: 2O04 A ∞ a 02) (Continued from p. 317) regulatory proteins caused a decrease in PC activation barriers. PS congenital defects, is also an autosomal dominant inheritance, lack of substantial liver injury and may also cause the PS to reduce V n1. PC, PS molecular defect is a congenital or acquired,[link widoczny dla zalogowanych], whether the anticoagulation can weaken the body of a physiological balance of coagulation, the coagulation hyperthyroidism, easy-to-2j】 【thrombosis. The experiments showed DVT in patients before treatment compared with the control group were significantly different (P 0.05). This shows that most patients with DVT are acquired PC, PS reduced, if the patient PC, PS activity determination results were lower before and after treatment should be considered as a congenital factors, to conduct family survey and genetic analysis. Clinical treatment of DVT, the majority of patients in the heparin therapy is usually the first or second day of oral anticoagulants,[link widoczny dla zalogowanych], which aims to suppress the four vitamin K dependent coagulation proteins (factor Ⅱ, Ⅶ, Ⅸ, X) synthesis, reduced protein C and protein S (anticoagulant protein) activation, and reduce their activity. Should also pay attention to the homozygous patients,[link widoczny dla zalogowanych], once the symptoms appear, must be given alternative therapy, including fresh frozen plasma or containing high concentrations of PC or PC prothrombin complex concentrate,[link widoczny dla zalogowanych], so that the original PC to keep the body level. At this point, laboratory PC, PS monitoring is essential.
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