Forum pracy nad projektem wskrzeszenia The Forge

[yoylqnqvwb]

CAH patients with calcium, phosphorus, and its clinical significance


Found in 1990 off the sixth meeting of the National Viral Hepatitis saying off the revised criteria,[link widoczny dla zalogowanych], 48 healthy volunteers as controls down. 1.2 The test method selected patients with morning fasting blood 2ml,[link widoczny dla zalogowanych], the serum was separated from Beijing kit put in a total of Biology Institute,[link widoczny dla zalogowanych], with the Netherlands produced VIA-TROISP - M Semi rash biochemical analyzer batch testing. Patients confirmed the test calcium, intravenous calcium gluconate lg / day or oral Gaitianli 300rag / day. Or combined with red Longmu Zhuanggu Song Jing and accounting treatment of pure VD / z one. f treatment. 2 results of chronic active hepatitis group and the control group serum calcium,[link widoczny dla zalogowanych], phosphorus results in Table 1,4 l chronic active hepatitis in the fall, l8 down lower than normal calcium group,[link widoczny dla zalogowanych], accounting for 43.87, down 13 higher than normal serum phosphate, accounted for 31.71 showed chronic active hepatitis group showed a low serum calcium 2.06 Soil 0.27 (P <0.01), hyperphosphatemia 1.40 +0.35 (P <0.01). Calcium and phosphorus concentration product 2.84. Lower than the control value of 2.90 calcium, phosphorus concentration decreased volume accounted for 51.22 hypocalcemia patients who were treated with calcium copper agents and VD, the Yao Tuiteng, liver health pain, insomnia, night sweats and other symptoms were significantly improved The results see the decline 2. Wrapped in a chronic active hepatitis group and the control group blood please calcium. Compare the decline of phosphorus concentration of calcium capsule 2 before and after the symptoms improved Park} t of calcium than the symptoms of lumbar pain in liver area boil Ju Qu night sweats fatigue symptoms of lumbar pain in liver area boil Ju Shin;; sweating weakness before treatment after treatment, the author of 41 Shiwei 3 Discussion Slow down people living liver calcium, phosphorus detection showed: 487 patients with hypocalcemia. 31.7l patients hyperphosphatemia, reduced serum calcium and phosphorus concentration product accounted for 51.22. Analysis of the low calcium causes: ① liver damage caused by 25-hydroxy vitamin Cable D (25 - OHD) decreased production ,25 - OHD formation in liver cells smooth endoplasmic reticulum, hepatitis patients with liver cells generally smooth endoplasmic reticulum damaged. 25 - OHD as calcium and phosphorus metabolism of important hormones, the shortage will reduce the intestinal absorption of calcium, blood calcium decreased bone deposition of calcium decreased. ② CAH patients with multiple poor appetite, long-term low heat, low protein, low calcium intake causes calcium decreased. Accustomed to low calcium diet plus China, Japan and for calcium is about 400mg, lower than developed countries 700 ~ 1300mg standards. Calcium reserve cars to be low, easily lead to the occurrence of bone disease. ③ Under normal circumstances, calcium, phosphorus concentration remained relatively balanced, calcium and phosphorus ratio of normal. The low calcium, high phosphorus and calcium. P ratio fell down, so that PTH secretion, resulting in bone loss, easily lead to the occurrence of bone disease in recent years, many studies confirm long-term sustainability of calcium, in addition to bone loss can be sent into, but can also cause blood vessels and other soft tissues calcium were increased. Cells containing the increase in calcium is considered to be the only way of cell death. Carbon tetrachloride induced acute hepatic necrosis 24 hours after the liver cells contain calcium significantly increased the degree of necrosis of liver cells and liver cells containing calcium is proportional to L When one is very low intracellular calcium, and high extracellular calcium, plasma membrane calcium concentration inside and outside the span of 5,000 to 10,000 times the full functionality of a cell - secretion, contraction, excitability, proliferation and secretion are determined by the transmembrane gradient of species distribution of calcium calcium deficiency liver disease patients according to the actual situation more obvious. I use the calcium therapy in such patients, the other with a calcium preparation and VD, but encourages patients taking calcium-rich foods such as milk, seaweed, soy products , shrimp, etc., all patients treated by the above-mentioned period, clinical symptoms were obviously improved. Therefore, in patients with chronic active hepatitis at the same time. trying to make up the calcium. is to promote the importance of liver disease early recovery methods.