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Adenosine echocardiography for evaluation of coronary artery disease


essechocardiographyfornoninvasivedetectionofcoro-naryarterydisease. JAmCotICardio [,[link widoczny dla zalogowanych],1994,74:1052-10565TawaCB. BakerWB,[link widoczny dla zalogowanych], KleimanNS, eta1. Comparisonofadenosineechocardiography. withorwithoutisometrichand · girp. Toexpertiseechocardiographyinthedetectionofis-chemiainpatientswithcoronaryarterydisease. JAmSocBchocardiogr. 1996.9:33-43 (200O3.o8 Received 2004-04-06 Revised) short brain-heart syndrome with case report of 76 cases Jing-Yi Wang Zhi new keywords acute cardiovascular disease, cerebrovascular accidents in recent years with acute cerebrovascular disease effects on the heart by the people's attention. Now our hospital because of acute cerebrovascular disease on brain heart syndrome reported as follows. I Materials and Methods 1.1 General information 一 2oo3 June 1999 in our hospital on Dec. 76 cases brain-heart syndrome, abnormal electrocardiogram,[link widoczny dla zalogowanych], or ECG,[link widoczny dla zalogowanych], based on the original appearance of new ECG changes. Are in line with brain-heart syndrome 28 cases, 48 ​​cases of conscious. electrocardiogram in cerebrovascular disease <7d there were 68 cases, ≥ 7d occurred in 8 patients. electrocardiogram abnormalities in 1 case continued two weeks, in 75 cases disappear within 7d. electrocardiogram changes in crown vein insufficiency the most common, followed by cardiac arrhythmia, myocardial infarction-like graphics to change, in which T changes in 44 cases, 13 cases of sinus tachycardia, paroxysmal supraventricular tachycardia in 6 cases, 6 cases of sinus bradycardia paroxysmal atrial fibrillation in 2 cases, 2 cases of supraventricular premature beats, kind of pseudo-graphic changes in myocardial infarction in 2 cases, 1 case of myocardial infarction in graphics, see Table 1. arrhythmias and ischemic changes in 19 cases of coexistence. 14 cases had hypokalemia or low chlorine exist hyperlipidemia, elevated enzyme in 39 cases, including alanine aminotransferase (GPT), aspartate transaminase (GOT), creatine kinase (CK), creatine kinase isoenzyme (CK-MB ), lactate dehydrogenase (LDH), lactate dehydrogenase isoenzyme (a-HBDH). 1.2 Treatment and prognosis has been given, vasodilators, improve coronary blood supply, application of cardiac, diuretic, protects the heart function, to correct hypokalemia, low chloride acidosis, timely use of antiarrhythmic drugs preferred potassium salt, and 8 of a blocker. 76 patients, except 1 case of disturbance of consciousness due to acute myocardial infarction who died, more than 75 patients (98.7%) conditions were better. Table 1 species of cerebrovascular disease and heart damage in patients (%) harm. manifested as changes in coronary insufficiency, mainly for ST segment and T wave changes, this group 57.89%; performance for a variety of arrhythmia, accounting for 38.16%; enzymes increased, accounting for 51.32%. Therefore, in patients with traumatic brain injury should be monitored ECG, long line of severe cardiac care, parallel enzyme examination, if the above scenario should take into account the disease, and to give timely treatment. aggressive treatment for primary disease, protect the heart, brain, kidney function, maintain normal water balance, add enough heat, if necessary, application of the arm to control arrhythmia, application of a potassium salt, and 8 blockers, can sometimes achieve better results. appropriate given diuretics to reduce cardiac stress, according to the different nature of cerebrovascular disease given vasodilators, thrombolysis and anti- coagulation and antispasmodic therapy. for those who have heart damage minimize the use of mannitol, because it can cause coronary artery spasm of coronary blood supply.