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Lisinopril on platelet activation in hypertension and insulin resistance in


Group by 4 weeks after treatment with lisinopril ,[link widoczny dla zalogowanych], compared with before treatment ,[link widoczny dla zalogowanych], blood pressure decreased significantly (PG Practical Diagnosis and Therapy 2003 17 4 0.05); IR significantly improved , ISI increased ( PG0.05). Buck also said明赖诺普利effective , can improve IR. ACEI is the drug class, has been reported to increase its ISI2O ~ 30. HIS mechanism may be reduced due to increased sympathetic nerve activity and adrenergic substances to reduce insulin resistance and improve insulin utilization. GMP 140 is a new discovery of a platelet membrane glycoprotein adhesion molecule Selection for the key members of the family , can be used as a specific marker for platelet activation . A 14O of hypertension in this group was significantly higher GMP , which is an important feature of MH [] . In the IR ,[link widoczny dla zalogowanych], the angiotensin Ⅱ (AT Ⅱ) activity increased, epinephrine also enhanced to promote platelet aggregation , platelet activation induced aggregation in the vascular endothelial damage at the release of TXA ,5-HT, microvascular spasm and thrombosis [1 ¨. Lisinopril blocking AT Ⅱ generation, and thus blocking platelet activation, adhesion molecule release. Improve the mechanism of IR is not entirely clear and may improve muscle tissue blood flow , reducing AT Ⅱ and norepinephrine concentrations , increase the release of bradykinin [7 can effectively prevent thrombosis. The study is a valid card明赖诺普利hypertension treatment of metabolic drugs. It can also correct the blood glucose blood pressure , platelet aggregation and other metabolic disorders.
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