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System identification method in the study of the cardiovascular regulatory function


), ..., £ (n +1 a p), Q; j a ~ (n4-I), A e (), ..., £ (n +2 a p)} I A e (n + N-1 ), a e (n + N-2), ..., a £ (n + N-p) C = (QQ) Q £ defined in (k) = e (q-) u (k), (k) = e (q A -) y (k) into the stone and on behalf of Y, x thus. Then 6 = (history Yao) a - Yao Y repeat the above calculation until 6 and convergence so far. We call the most. For the steady-state gain. In addition, we use the FFT method the original data {Y) of the power spectrum S (k), kffi0, I, 2, ..., ITI. Of S (k) a simple mathematical transformation, so p, k-o, 1,2, ..., mΣs (j) and make it meet the p ≥ 0, ΣPf1 definition of entropy H = a ΣPlogp we can entropy of the steady-state gain K and H to both the amount of analysis and evaluation of free-to-Speech Festival cardiovascular system quality. The results are shown in Table I and Table 2. Results and discussion Huan l ● apricot gain K changes sac sac 2gll1) f ~ iTitb1e1Theehaag ~ ofsIe-d, Btate ~ (tlnKTable2Theeh4nEe. Fentropy'41ue Aerospace Science and Medical Engineering and even see the reserpine group before treatment than after treatment significantly increased K; atropine group smaller steady-state gain K; the control group changed little. reserpine groups can be seen from Table 2, the value of information entropy have a greater change in H . injection of reserpine, the blood pressure regulating quality development to the non-steady state, H value of 5.386 from the increased to 6.056 prior to injection, which reflects the degree of ordering within the system to change. reserpine sympathetic nervous system interference 】 normal functional status, the system is disturbed, the original organizational capacity, regulatory mechanisms fail, allowing the system to the non-binding, non-movement direction of the organization, leading to H becomes larger. injection of reserpine, the negative in a 30mmHg pressure, rabbit endurance, heart rate, mean arterial pressure was significantly lower (p 005). This shows that blood pressure remained essentially the original system ordered. controls the steady-state gain K and the information entropy H of two experiments on the changes before and after are not large, no statistically significant (P> O.05). According to pharmacological knowledge, reserpine is the role of the sympathetic impulse conduction delay, lower blood pressure, heart rate . atropine is to remove the vagus nerve on cardiac arrest suppressed, so that the heart suspect accelerated; improve the microvascular circulation. nerve on cardiovascular regulatory function 0】: vagus nerve heart, rustic rooms node atrioventricular node and ventricular conduction system. vagus Fen , heart rate; vagal inhibition of heart mind and direct pressure of 3 cents cavitation T = ble3Theck = ngeB ● fHRa = dBP rubidium stronger role. sympathetic innervation in various parts of the heart, including ventricular muscle. sympathetic activation can increase blood pressure to enhance myocardial contractile force and accelerate myocardial metabolism, leading to vasodilators generation, increase coronary blood flow. parasympathetic and sympathetic dominance of the heart to maintain cardiac function in balance and stability, the need to adapt to environmental change. Our experimental results and theoretical is consistent with Taiwan. The results show that the pressure under who are charged with regulating the cardiovascular function, or K steady-state gain information entropy H to quantitatively characterized. Where under negative pressure, or the steady-state gain K larger entropy values ​​of H are poor cardiovascular regulatory function. On the contrary, regulatory function is good.