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Gastric lesions of portal hypertension


The detection rate is similar to the control group (P> 0.05). Gastric diffuse liver disease group, but edema (with or without multiple moose bad) in 32 down. Accounted for 26.23%. While the control group only 2 down. Accounted for 1.64%,[link widoczny dla zalogowanych], both with significant difference (P 0. (15) which showed that gastric diffuse congestion and edema. Fundus, body erosion of the stomach at the bottom of Lin-like congestion spots when portal hypertension is a common stomach damage performance . have considered the formation of portal pressure and is closely related to the degree of esophageal varices. because of disease or bypass of Sri Lanka sclerotherapy varicose vein is invalid. and can be increased. The split wood and reduce portal pressure drugs can improve disease ... . have information to report, Xi Fang laser multi-detection blood Otaki gastric mucosal blood flow in patients with liver cirrhosis. The results showed that the gastric mucosal blood flow than the control group J. electron microscopy, scattered a significant expansion of mucosal capillaries, capillary significantly increased vascular holes, widened uJ. these further instructions as the impact of portal hypertension. venous obstruction. capillary pressure increased. circulatory disturbance caused by gastric mucosa. mucosa barrier damage,[link widoczny dla zalogowanych], resulting in gastric mucosal hyperemia and edema and moose bad as when the bleeding portal hypertension basic lesions. the mechanism of peptic ulcer patients with liver cirrhosis is not clear. has weakened that defense function of gastric mucosa may be its main factors. because most patients with liver cirrhosis the basis of stomach acid is low, with a group of five belly Fu {amine or more stimulates the stomach after the MAO half the people are still lower than normal. Upon information 122 hot down the door of liver cirrhosis in patients with hypertension, digestive endoscopy detected ulcer in 19 cases. detection rate of 15.57%. and the reported 5% ~ 15% _ | similar, and the control group compared with the detection rate of 23.77%, no significant difference. but dead solution data, liver cirrhosis with portal hypertension group the detection rate of non-ulcer non-cirrhotic portal hypertension group than double. Upon information and autopsy data are inconsistent, indicating cirrhosis in patients with increased risk of ulcer disease after a hard The risk factors are not hills. In addition, it is worth mentioning that the clinical and endoscopic ulcer disease diagnosis rate,[link widoczny dla zalogowanych], portal hypertension was significantly lower than the control group, and clinical diagnosis of cirrhosis and portal hypertension group 3 down, 2 cases because of the history of the black keys will be prompt. only 1 down Park abdominal cancer was considered. cirrhotic patients showed typical symptoms of peptic ulcer, or masked by symptoms of liver disease. To easily Lan clinical diagnosis of gastric cancer in liver disease not detected a fall in the control group were detected in 5 cases. because, although non-liver disease control group were non-portal hypertension. but have different levels of the slander of the tract symptoms. it is not good to make statistical conclusions. the same with or without { Gan about because of tangled,[link widoczny dla zalogowanych], pending further observation. 4