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Metoprolol in chronic severe heart failure of


F to obtain good results. In view of the group room rate of CHF patients with multiple associated with chronic atrial fibrillation or sustained growth rate of persistent sinus tachycardia, their treatment is effective or not, depends largely on the growth of the ventricular rate can be effectively controlled. The addition of metoprolol group, the various indices were significantly slower heart rate, cardiac function and activities at the same time have been better and improve endurance. In the past that if compensatory sympathetic excitation contraction of heart failure may improve, so that to maintain the hemodynamics in patients with heart failure and stroke volume. However, recent studies have shown that high levels of catecholamines does not make praise be useful depleted myocardial effect but instead may lead to reduced myocardial receptor density B, which will lead to failure of myocardial function has been further deterioration. And 0-blockers, there sympathetic inhibition and reduced adverse effects caused by shock,[link widoczny dla zalogowanych], then. Although treatment of B-blocker CHF has not finished the exact machine translation barrier clear, but at least the following beneficial effects: (1) CHF tea when the excessive release of several phenolic amine depletion of myocardial energy storage, Results citrate change, functional disorders, B blockers to protect against such direct myocardial damage and reverse the above pathological damage may t (z) l53 · severe CHF due to chronic long-term sympathetic activation induced myocardial B receptor density decreased, thus changing force and lack of response to chronotropic stimulation, B blockers can increase cardiac B receptor density and restore the heart's sympathetic supporting role on the failure to cut off the vicious cycle t (3) CHF, including when the power increase can be over-sympathetic stimulate the renin-angiotensin-aldosterone system one, leading to a series of hemodynamic and renal excretion function disorders, at the neuroendocrine blockers can block the way; (4) B blocker with slow heart rate,[link widoczny dla zalogowanych], reduce myocardial tension, improve their Mian should be of (diastolic function) and reduce the role of myocardial oxygen consumption based on the above we believe that even though B receptor blockers on cardiac negative inotropic effect though, but in the heart of chronic severe CHF who there is a wide range of protection, so the net effect is still beneficial to the repair of CHF and reversed. Given the experience of B blockers CHF is still very limited,[link widoczny dla zalogowanych], individual differences in sensitivity and tolerance is very clear,[link widoczny dla zalogowanych], must be very careful when using,[link widoczny dla zalogowanych], note the following points: (1) B-blockers is mainly used for relaxation to be of poor decreased Mian CHF and severe CHF associated with chronic supraventricular arrhythmia with rapid sinus rhythm and those with rapid t (2) dose should be small, increase the amount to be slower, long-term aim is to allow the medication has enough heart failure time increased the number of receptor t (3) be sure to benefit with feces and other cardiac therapy combined with conventional anti-heart failure, and should be used in day. Blockers is due. Any choice of treatment are required for each specific patient under a variety of practical factors to consider its eight barrier face disadvantages may be the choice.